SUP3R-SHRED by Olympus UK
Serving Size: 2 caps
Servings per Container 30
Artemesia Lwayomogi Extract 30mg
Introducing SUP3R-SHRED, where science and shredding meet!
We at Olympus UK are proud to present a compound never before brought to market! A leap forward in innovation, Alpha tested in house for safety and efficacy, we bring you… SUP3R-SHRED!
So, what is the active ingredient in SUP3R-SHRED that makes it innovative? Well, SUP3R-SHRED contains Dibromo-4-methoxybiphenyl, a naturally occurring molecule derived from the herb Artemisia Iwayomogi (of the Asteraceae family), commonly referred to as “Haninjin,” one of the major medicinal materials used in traditional Korean medicine, which can also can be found in Korean cuisine.
Data regarding a 95% ethanolic extract of Artemisia Iwayomogi, which contained 3.55% Dibromo-4-methoxybiphenyl, showed the ability to activate PPAR-Delta (peroxisome proliferator-activated receptor delta), a mechanism that is unique among supplements. This same extract was shown to increase skeletal muscle fatty acid oxidation and increase the expression of genes involved in lipid catabolism, as well as attenuate body weight gain in high fat diet-fed mice and thus protect against diet-induced obesity. It is plausible that Dibromo-4-methoxybiphenyl alone is directly responsible for this effect, as it does share a weak structural similarity to GW501516 (Cardarine), a synthetic PPAR-Delta agonist noted for similar effects. However, unlike GW501516, Dibromo-4-methoxybiphenyl is a naturally occurring molecule with a long-standing safety record as a component of Haninjin, which has been consumed in Korea for generations as part of traditional Korean medicine. This makes Dibromo-4-methoxybiphenyl the safest and strongest PPAR-Delta agonist on the market!
So what is PPAR-delta, and what does agonism (activation) of this receptor do? Simply put, PPAR-Delta is a regulator of lipid metabolism, and agonism of this receptor has been shown to induce fatty acid oxidation via PGC-1alpha. In fact, it changes the body’s fuel preference from glucose to lipids. Brunmair et al. reported that PPAR-Delta activation acts to suppress glucose utilization as a result of a switch in substrate preference from carbohydrates to lipids in skeletal muscle.  That translates to enhanced body fat loss and improved endurance capabilities. And may even potentially lead to enhanced recovery, as PPAR-Delta activation is also associated with enhanced skeletal muscle regeneration. PPAR-Delta activators even have positive general health effects, such as being shown to improve insulin resistance, reduce plasma glucose and reduce serum triglycerides
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